Download Basic Guide to Anatomy and Physiology for Dental Care by Carole Hollins PDF

By Carole Hollins

Content material:
Chapter 1 easy Biology – evaluation (pages 1–19): Carole Hollins
Chapter 2 Cardiovascular method (pages 20–39): Carole Hollins
Chapter three respiration approach (pages 40–52): Carole Hollins
Chapter four Digestive method (pages 53–74): Carole Hollins
Chapter five anxious approach (pages 75–87): Carole Hollins
Chapter 6 Oral Embryology and Histology (pages 88–106): Carole Hollins
Chapter 7 cranium and Oral Anatomy (pages 107–141): Carole Hollins
Chapter eight the teeth Anatomy (pages 142–157): Carole Hollins
Chapter nine Periodontal Anatomy (pages 158–166): Carole Hollins
Chapter 10 Salivary Glands (pages 167–173): Carole Hollins

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Additional resources for Basic Guide to Anatomy and Physiology for Dental Care Professionals

Example text

Crucially, the reduced blood flow to the kidneys results in fluid retention, and this excess fluid tends to accumulate in the body tissues and is called oedema. When the right side of the heart is failing (right-sided heart failure), the excess fluid will pool in the body peripheries, especially the ankles, and is referred to as peripheral oedema. A more serious situation occurs when the left side of the heart is failing (leftsided heart failure), as the fluid accumulation occurs in the lungs themselves.

Capillaries have the following features: • Endothelium – made up of just a single layer of cells, so that gases, nutrients and waste products can pass across them easily in an exchange mechanism • No muscle layer – this would prevent the exchange mechanism from occurring • No elastic layer – this is not required as the blood flow in these vessels is no longer pulsatile, and again, it would prevent the exchange mechanism from occurring It is in the capillary beds that the oxygen inspired in the lungs passes out of the circulatory system and into the body tissues to be used to create energy by the cells.

Emphysema A disease caused by pollutant damage to the alveolar sacs and the alveoli themselves, resulting in their distension (abnormal widening). The main pollutant is tobacco smoke, although industrial pollutants also contribute to the damage caused. The presence of these pollutants in inhaled air causes the alveoli to release chemicals that damage their walls and the lung parenchyma, and over time the damage can be so great that the alveoli eventually burst, and the lung structure loses its elasticity.

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